A type 1 diabetic mystery is why do some Type 1s get complications and others seem to never get them? A massive Japanese study of Type 1 diabetics found that those with fulminant diabetes developed complications much faster and more severely than those with non-fulminant diabetes.

The difference between fulminant and non-fulminant is the speed and intensity at which the disease develops. Fulminant Type 1 diabetes typically develops suddenly with near total loss of beta cell function. This type of diabetes is confirmed with testing c-peptide levels. Non-fulminant type 1 diabetes has residual c-peptide levels that eventually taper to undetectable. Sometimes this is seen through many years of the Honeymoon Period.

This study may be the antithesis of conventional wisdom for preventing complications. Staking all hopes on blood sugar control is heavily optimistic. Yes controlling blood sugar does lessen the workload for existing beta cells, and thus extends the lifespan of each beta cell. Research suggests that c-peptide offers protection to beta cells, both from apoptosis (cell death) and encourages new cell growth. This new cell growth applies to beta cells and other cells of the body that endure long-term Type 1 diabetes complications.

Diabetics are instructed that maintaining normal blood sugars is the Holy Grail of preventing long-term complications. Yes and no. The truth is controlling your blood sugar will not allow complications of Type 1 diabetes to develop as quickly, presuming you still had some level of beta cell function upon diagnosis (i.e., c-peptide). That doesn't sound like a reward as much as it does a delayed punishment. I'd like c-peptide with my insulin, please. It's off the à la carte menu? That's fine - serve it up! I want to thank Klausen for bringing this study to my attention.

Creator of Diabetes Health Magazine, Scott King, has been a type 1 diabetic for over 34 years. Needless to say, he knows diabetes, and he is doing a remarkable job of introducing cutting-edge treatments for diabetics. In the first Diabetes Health TV broadcast, he shared interviews from the recent AADE Conference. A really exciting product he featured is called Neuragen - a topical treatment for diabetes neuropathy.

With diabetes neuropathy, people experience pain due to damage to the peripheral nerves. Neuropathic pain is often characterized by burning sensations or shooting pain, or may occur as numbness or chronic itching. Clinical trials have shown Neuragen to be effective in 70% of patients for the pain associated with diabetes. The ingredients are pretty kosher, too. Neuragen is made of a proprietary blend of essential oils from special species of geranium, lavender, bergamot, eucalyptus, and tea tree.

The Neuragen rep was blunt when he described the effective nature of this all natural product - using more does not make it any more effective! You have to admire his refreshing honesty. But like I said upfront - if Scott King is willing to spend the time getting the scoop on this product - it's probably worth your time using it. For more interviews, checkot the full coverage of the AADE Conference on Diabetes Health TV!

Now that the US market is suspiciously saturated with human insulin - and many of us diagnosed within the last 10 years did not have a shot at trying porcine insulin - I'd like to set the record straight. When the pharmaceutical companies cherry pick the studies they wish to use for their gain, and not so much for your enhanced quality of life - they must've lost this study.

Please read the entire study (if you have access to it in a local library) but what grabbed my undivided attention was the sentence that says: it was observed that the action of porcine insulin was associated with... a striking increase of prolactinaemia, in relation to semisynthetic human insulin.

Okay -- so as I look deeper into the function of prolactin -- aside from some definite dopamine enhancing activities (if you know what I mean) :::wink wink::: -- it is responsible for the formation of myelin coatings on axons in the central nervous system. This is a certifiable problem that results in diabetic neuropathy and the related side effects (numbness, nerve dysfunction, i.e, ED).

Ex-queeze me? Does this say that human synthetic insulin may be a cock blocking drug?

Sorry for the blunt delivery -- but this is the truth. Why doesn't human synthetic insulin have this listed as a side effect? My guess is: if you had a choice of human synthetic insulin versus highly purified porcine insulin -- and you knew the side effects of human synthetic might take a toll on the health of your sex life -- you might be praying to the porcine gods.

Shame on the companies who knew about this study and kept it undercover so you couldn't...

Diabetes Care reported the size of the spinal cord is significantly diminished long before symptoms of nerve damage appear in adults with diabetes. The study found that MRI assessment of the spinal cord may be helpful for detecting silent signs of spinal nerve damage in patients with diabetes.

Diabetes can lead to nerve damage or "diabetic neuropathy" -- a painful condition that causes a range of symptoms from a tingling sensation or numbness in the toes and fingers to paralysis. Doctors studied 84 men with type 1 or insulin-dependent diabetes, 24 nondiabetic controls and eight subjects with an inherited neuropathy. Nineteen of the diabetic subjects had no diabetic neuropathy, 23 had silent or "subclinical" neuropathy and 39 had clinically detectable neuropathy. MRI (magnetic resonance imaging) of the spine showed that the spinal cord area, corrected for age, height and weight, was 67.5 mm in diabetics without neuropathy, 62.4 mm in diabetics with subclinical neuropathy and 57.2 mm in diabetics with overt neuropathy. There were no significant differences in the spinal cord area of diabetics without neuropathy and nondiabetic controls.

Spinal shrinkage may be a silent sign of diabetic neuropathy - but we're not taking this lying down. We hear you, loud and clear! Those of you degenerate attributes of diabetic neuropathy who think you can go about your business undetected - you've got another thing coming. Watch your back. Modern medicine is coming up with impressive treatments and we're gonna come at you like a spider monkey!

A study from U.S. researchers has found that daily supplementation with a cinnamon extract may boost the body's antioxidants, which can lessen the complications associated with metabolic syndrome. Metabolic syndrome is characterized by obesity -- especially around the midsection -- hypertension and reduced metabolism of both glucose and insulin. The syndrome is associated with an increased risk in the development of type 2 diabetes and cardiovascular disease.

The study recruited 24 participants with impaired fasting glucose levels for a double-blind, placebo-controlled study. The patients were split into two randomly assigned groups: the first received a daily dose of 500 mg of cinnamon extract and the second received a placebo. After 12 weeks, the researchers found that in the cinnamon extract group, antioxidant levels were significantly increased compared to the placebo group. Plasma levels of a reactive compound related to oxidative stress were also lower in the cinnamon group, but remained unchanged in the placebo patients. Oxidative stress is responsible for many of the microvascular changed responsible for diabetes complications: blindness, numbness, kidney damage, and amputations.

Beyond the resounding proof of cinnamon being helpful in diabetes management, other health-related benefits include: improved digestion, toning of tissues, relief from congestion, muscle and joint pain relief, relief from menstrual cramping, thinning of the blood and better circulation, relief from arthritis pain, prevention of urinary tract infections, prevention of tooth decay, and killing of harmful bacteria. I think a sprinkle of cinnamon is worth the pound of prevention this little spice is punching. How much is 500 mg of cinnamon anyway?