Now that the US market is suspiciously saturated with human insulin - and many of us diagnosed within the last 10 years did not have a shot at trying porcine insulin - I'd like to set the record straight. When the pharmaceutical companies cherry pick the studies they wish to use for their gain, and not so much for your enhanced quality of life - they must've lost this study.

Please read the entire study (if you have access to it in a local library) but what grabbed my undivided attention was the sentence that says: it was observed that the action of porcine insulin was associated with... a striking increase of prolactinaemia, in relation to semisynthetic human insulin.

Okay -- so as I look deeper into the function of prolactin -- aside from some definite dopamine enhancing activities (if you know what I mean) :::wink wink::: -- it is responsible for the formation of myelin coatings on axons in the central nervous system. This is a certifiable problem that results in diabetic neuropathy and the related side effects (numbness, nerve dysfunction, i.e, ED).

Ex-queeze me? Does this say that human synthetic insulin may be a cock blocking drug?

Sorry for the blunt delivery -- but this is the truth. Why doesn't human synthetic insulin have this listed as a side effect? My guess is: if you had a choice of human synthetic insulin versus highly purified porcine insulin -- and you knew the side effects of human synthetic might take a toll on the health of your sex life -- you might be praying to the porcine gods.

Shame on the companies who knew about this study and kept it undercover so you couldn't...

A study shows that men with type 2 diabetes experience pronounced risk of erectile dysfunction (ED) when they have symptoms of depression.

Depression appears to be a vicious cycle that may instigate the development of ED, while the ED symptoms perpetuate the symptoms of depression. Effectively deducing that ED in diabetic patients is not related entirely to organic factors. The study evaluated men with type 2 diabetes who completed questionnaires every 6 months for 3 years. Five hundred men reported ED at the start of the study, and an additional 192 developed the disorder during follow-up. The investigators noted higher prevalence of high blood pressure (46% versus 32%) and lipid abnormalities (23% versus 13%) among men who complained of ED. The researchers also observed higher incidence of retinopathy, neuropathy or heart and vascular disease in ED sufferers. Those with ED were more likely to be treated with insulin or diuretics. Depression and ED was found more often in those with poor physical and psychological health.

The researchers surmised that erectile problems are not necessarily an inevitable outcome of the aging process. The risk factors for ED are modifiable and can be changed to reduce the likelihood of the outcome (or lack thereof). The results should serve as uplifting news for suffers of ED, no pun intended.